Max in WT synaptoneurosomes, suggesting that Src signaling may very well be downregulated in KI synapses. On the other hand, our ability to rescue SERT function in KI midbrain synaptoneurosomes from the inhibition of FAK implies elevated FAK signaling downstream of your Pro32Pro33 mutant, as verified by enhanced pFAK localization https://peterk531pal3.wikiexpression.com/user